Acute Kidney Injury (AKI) is a common and serious complication in
patients who have experienced cardiac arrest. It results from a complex
interplay of ischemia-reperfusion injury, hemodynamic instability, and systemic
inflammatory responses, all of which contribute to renal dysfunction. AKI is
associated with increased morbidity and mortality, prolonged hospital stays,
and higher rates of long-term renal impairment. Targeted temperature
management (TTM) has been widely implemented as a neuroprotective
strategy following cardiac arrest, aiming to improve neurological outcomes and
overall survival. However, its impact on kidney function remains an area of
active investigation. Understanding the effects of TTM on AKI development,
severity, and recovery is crucial for optimizing post-cardiac arrest care.
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