Copper, an essential trace element, plays a pivotal role in various biological processes. However, excess copper levels can lead to cytotoxicity, causing oxidative stress and cell damage. One crucial determinant of copper-induced cytotoxicity is its reduction potential, influencing its redox activity within cells. This article delves into the intricate relationship between copper reduction potential and reductive cytotoxicity, exploring underlying mechanisms, implications for health and potential therapeutic interventions.
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