Remyelination is the process by which precursor cells of oligodendrocytes are propagated to form oligodendrocytes to create new myelin sheaths on demyelinated axons in CNS. It is a mechanism that is controlled naturally within the body and appears to be very successful in a stable CNS. The process produces a thinner sheath of myelin than normal, but it helps protect the axon from further injury, from overall degeneration, and once again proves to increase conductance. The underlying remyelination mechanisms are being studied in the hope of discovering therapies for Demyelinating disorders, such as multiple sclerosis. Remyelination is triggered and controlled by a variety of factors that control the migration and differentiation of Oligodendrocyte Precursor Cells around the lesion sites. Remyelination in the structure of the developed myelin appears different from developmental myelination. Reasons for this are uncertain but the axon 's proper role is restored regardless of.
Extended Abstract: Mental Disorders and Treatment
Extended Abstract: Mental Disorders and Treatment
Review Article: Mental Disorders and Treatment
Review Article: Mental Disorders and Treatment
Short Communication: Mental Disorders and Treatment
Short Communication: Mental Disorders and Treatment
Research Article: Mental Disorders and Treatment
Research Article: Mental Disorders and Treatment
Research Article: Mental Disorders and Treatment
Research Article: Mental Disorders and Treatment
Posters & Accepted Abstracts: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Keynote: Neurological Disorders
Keynote: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Keynote: Neurological Disorders
Keynote: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
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