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Pathogenesis of alimentary diabetes: recovery by loss of 20% body weight, and by attainment of initial hunger as well as of low BG before meals
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Medicinal Chemistry

ISSN: 2161-0444

Open Access

Pathogenesis of alimentary diabetes: recovery by loss of 20% body weight, and by attainment of initial hunger as well as of low BG before meals


6th World Congress on Medicinal Chemistry and Drug Design

June 07-08, 2017 Milan, Italy

Gaia Cecchia

Nutrition & Prevention, Italy

Scientific Tracks Abstracts: Med Chem (Los Angeles)

Abstract :

Background & Objective: We attempted to train two diabetic adults as we suggested in the first abstract. Diabetic people are different from healthy people in this: they donâ��t develop any hunger sensation after meal suspension. Methods: Training: recognizing Hinitial Hunger and associate this sensation to low Blood Glucose (76.6 �± 3.7 mg/dL). We tried to implement this training in two obese, diabetic adults.The two subjects consumed meals devoid of fats and carbohydrates (Very Low Energy Diet, VLED) for 6 to 12 months. Results: At recruitment the two diabetic subjects (out of two) showed a BMI of 39 and 33 and they neither developed a BG decline to 76.6 �± 3.7 mg/dL nor any hunger sensation after 2-days eating suspension. Then Both subjects lost 13%-20% of their initial body weight; they recovered 76.6 �± 3.7 mg/dL of BG and hunger sensations before one â�� three meals a day, i.e.: they went out of diabetes. Conclusion: Diabetes develops by inveterate conditioned intake (when previous energy intake has not been fully exhausted before meals), excessive fattening, excessive post-absorption emission of fatty acids from fatty tissues, permanent loss of BG decline to 76.6 �± 3.7 mg/dL and permanent loss of physiological signals of hunger. A healthy, non-diabetic life may be recovered by a painless loss of 20% body weight (No fats, no carbohydrates) and may be maintained by implementing IHMP at reappearance of hunger sensations. This means accurate energy intake planning instead of hunger endurance.

Biography :

Gaia Cecchia has done a course in Mathematics from the University of Florence. She graduated in Pharmaceutical Sciences in the year 2012 with a thesis in neuropharmacology with the title: “Memantine and topiramate in association with hypothermia as neuroprotective agents in the neonatal ischemic encephalopathy”. Since 2012, she is working at the ONLUS-No-profit association ‘Nutrition&Prevention’ with Professor Mario Ciampolini.

Email: gaiacecchi280@gmail.com

Google Scholar citation report
Citations: 6627

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